Cervical Dystonia is painful and inconvenient – how can it be made more tolerable for the person suffering from it?
While there is no cure for this disorder, SPG blocks can be extremely helpful in managing Cervical Dystonia. Let’s observe how a certain patient has reacted to self-administering SPG blocks and how it has changed her experience with chronic pain.
What Are SPG Blocks?
First, let’s talk about what SPG blocks are, and how they are useful in this situation.
SPG stands for Sphenopalatine Ganglion, and refers to a group of nerves that are found behind the bone structure of your nose. This bundle of nerve cells is linked to the trigeminal nerve, which is the primary aspect involved in headache disorders.
The procedure essentially works by numbing your nose and having a catheter device pushed up one of your nostrils. After the SPG is numbed, the process is repeated through your second nostril.
SPG blocks are safe to use and patients can actually self-administer them in their own homes for less than $1 per application. The side effects are nothing to fear either- they include decreased anxiety and depression, better blood pressure, a reduction in sympathetic overload, and less stress.
How SPG Blocks Can Change the Cervical Dystonia Experience
One patient suffering from Cervical Dystonia has been self-administering SPG for one month to treat her chronic pain and has seen fantastic results.
During her first visit for discussing treatments for the Cervical Dystonia, she had a consultation and was given some easy exercises to complete. At first, she did have some big concerns about being able to administer the SPG blocks herself but quickly picked it up successfully.
Now she is able to administer the SPG blocks daily and complete the exercises at home without any issues. Here is her video testimonial where she shares how different and improved her quality of life is now.
Utilizing SPG Blocks with Cervical Dystonia
Chronic pain is something that can seriously affect the happiness and quality of life, so it is vitally important to find a solution that works for each patient. For those suffering from Cervical Dystonia, it is highly recommended to reach out to a local doctor for further information on utilizing SPG blocks for chronic pain.
If located in the Chicago/Milwaukee area, Dr. Shapira normally practices in Gurnee and Highland Park. However, he also sees some national and international patients as well.
Anxiety and Stress problems are very common among patients with TMJ disorders. \II have trained many neuromuscular dentists in the use of Sphenopalatine Ganglion Blocks.
Sympathetic overload is common in a host of medical issues made worse by stress. The Sympathetic nervous system controls the “Fight or Flight” reflex. While this reflex can be a lifesaver in some dangerous circumstances it can often become destructive when it goes into overdrive and causes Phobias and Social Anxiety.
The Sphenopalatine Ganglion (SPG) is the largest Parasympathetic Ganglion of the cranium but just as important for patients with phobias and social anxiety is the Sympathetic nerves of the Superior Sympathetic Chain also pass thru the ganglion.
Social Anxiety Disorder (SAD) is a condition where “normal” social interactions cause severe and seemingly irrational anxiety. This disorder can last for years or even an entire lifetime. Social Anxiety Disorder may seem irrational but may have originally been triggered by a single event or series of events where the Sympathetic nervous system triggered changes in the victim’s brain that repeatedly cause the Sympathetic Nervous System to act explosively during everyday social interactions resulting in irrational anxiety, fear, embarrassment, and extreme self-consciousness.
Social Anxiety like other phobias can be a learned response that appears to be irrational when the original triggers are forgotten or repressed by the brain. Post Traumatic Stress Disorder (PTSD). Social anxiety and Social Anxiety Disorder differ from PTSD because the memory of the precipitating events is lost but the reaction of the autonomic nervous system still responds to those forgotten or hidden memories. Sufferers exhibit avoidance of situations that bring back autonomic symptoms experienced from the trauma.
The physical and mental responses have a primarily autonomic nervous system foundation. Turning off the autonomic “Fight or Flight” Sympathetic reflex can be life-changing. Many medications have been utilized over the years to help patients deal with these symptoms. Propanolol (Inderal) is a Beta Blocker that has been utilized to help people overcome this reaction.
Sphenopalatine Ganglion Blocks are utilized for treating a wide spectrum of pain disorders. Originally described in 1909 by Greenfield Sluder for treating Sluder’s Neuralgia (sphenopalatine neuralgia). The Sphenopalatine (Pterygopalatine) Ganglion Block was the subject of a 1930 article, “SPHENOPALATIINE PHENOMENA” in the Annals of Internal Medicine (JAMA) BY Hiram Byrd MD which looked at 10,000 blocks in 2000 patients for a wide variety of medical disorders.
Greenfield Sluder later wrote a medical textbook “Nasal Neurology” which covered the topic in great detail. Unfortunately, the pharmaceutical age arrived and this amazing block became part of “Forgotten Medicine” had it not been for a 1986 popular book “Miracles on Park Avenue” which detailed the medical practice of Dr Milton Reder an octogenarian ENT in New York City who treated thousands of patients from around the world with only Sphenopalatine Ganglion (SPG) Blocks.
I have been utilizing these “Miracle Blocks” since 1986 after a patient gave me a copy of that book and asked me to find someone in Chicago who did SPG Blocks. There were no doctors anywhere in Illinois or Wisconsin utilizing these blocks but a friend who also treated TMJ disorders, Dr Jack Haden in Kansas City knew how to perform SPG blocks and I learned the technique from him.
I initially utilized the blocks to treat patients in my office in a manner similar to Dr Milton Reder but eventually began to teach my patients how to self administer the blocks with cotton-tipped nasal catheters. Originally, I utilized a 10% cocaine solution but switched to lidocaine over 30 years ago. Lidocaine was extremely safe and made self-administration practical.
Self-Administration is key to successful use of SPG Blocks for treating phobias and Social Anxiety because it puts the patient in control or turning off the “Fight or Flight” Sympathetic reflex and turning on the Parasympathetic reflexes, the ‘Feed and Breed” or “Eat and Digest” which turn off stress and turn on feelings of safety and calm. They are the same responses we feel when playing with puppies, kittens or babies.
SPG Blocks are considered a first-line treatment for Cluster headaches but are utilized for a wide variety of chronic headaches especially Trigeminal Autonomic Cephalgias, Tension Headaches, Chronic migraines of all types, Chronic eye pain, ear pain, sinus pain and throat pain. Recent studies have shown SPG Blocks can cure about 1/3 of essential hypertension cases. Cranio Journal will publish a new article I authored in it’s May 2019 Journal issue.
Treatment of PTSD, Phobias and Social Anxiety is multifaceted but Self-Administered SPG Blocks as an integrated part of the treatment will probably become a Standard of Care in the future.
The Stellate Ganglion (SGB) Block has been called “The God Block” and when used for shoulder pain has been shown to “Cure PTSD” with a single shot. The military is currently doing a multi-million dollar study to evaluate Stellate Ganglion Blocks for treating PTSD. The Stellate Gangliois a large Sympathetic Ganglion in the neck. It is also part of the Autonomic nervous system.
This website has the most extensive review of all published material on the subject of Sphenopalatine Ganglion Blocks available in a series of Blog posts.
The trigeminal cardiac reflex (TCR) is a unique, powerful, and well-established neurocardiogenic reflex that is a result of stimulation along the path of the fifth cranial nerve (trigeminal nerve). It can produce adverse cardiorespiratory changes including hypotension, bradycardia, and asystole, as well as gastric consequences such as hypermotility. This reflex has been reported to occur in various surgical conditions, as well as in neurosurgical interventions.
Sleep bruxism, thought to be a more intense form of rhythmic masticatory muscle activity, has a prevalence of about 8%1 and has been explicitly linked to the TCR.2We report a case of a young woman with severe bruxism who incited her TCR, which subsequently produced profound nocturnal pauses that ultimately required dual-chamber pacemaker implantation.
A 27-year-old woman presented with palpitations and syncope. Three years prior to presentation she developed nocturnal and early morning nausea and vomiting that would often wake her from sleep. She was noted to have a long-standing history of severe bruxism with physical signs on examination of significant attrition. This had persisted despite the use of a retainer and bite block. Evaluation with Holter monitoring revealed sinus bradycardia, intermittent second-degree type II atrioventricular (AV) block, and a pause of 8.6 seconds (Figure 1). Interestingly, the rhythm strips showed simultaneous effects on both the sinus and AV node, suggesting an autonomic etiology. Of note, these rhythm disturbances were principally nocturnal in nature. While she was wearing the Holter, the husband was awake and corroborated that she was having severe episodes of bruxism. Further cardiac evaluation was unrevealing, including a normal echocardiogram, cardiac magnetic resonance imaging, sleep study, and thorough autonomic testing. With her constellation of symptoms—severe bruxism, AV nodal block with cardiac pauses (that were predominantly nocturnal), and gastrointestinal symptoms—we diagnosed her with hypervagotonia from stimulation of the powerful TCR from severe bruxism (Figure 2). Out of concern for risk of cardiac death from these pauses without a stable ventricular escape, we elected to place a dual-chamber pacemaker for bradycardic prevention.
This case highlights the intricate and noteworthy relationship between the autonomic nervous system and the heart. Our patient developed high-grade AV block and syncope owing to significant and profound hypervagotonia. Based upon her evaluation and corroboration of these events by her husband, we deemed that her intense vagal stimulation was a consequence of her severe bruxism, which was eliciting the TCR.
The TCR (Figure 2) is a powerful brain stem reflex that can be associated with a sudden onset of parasympathetic dysrhythmia, sympathetic hypotension, or gastric hypermotility. The proposed mechanism of this reflex is stimulation of the sensory nerve endings of the trigeminal nerve (Figure 2, cranial nerve V), which sends signals via the Gasserian ganglion (Figure 2, indicated by asterisk) to the sensory nucleus of the trigeminal nerve (Figure 2 inset). The afferent pathway then continues along the short internuncial nerve fibers in the reticular formation to connect with the efferent pathway, the motor nucleus of the vagus nerve (Figure 2, cranial nerve X). The last part of the reflex is formed by cardioinhibitory efferent fibers, which connect the motor nucleus of the vagus nerve to the myocardium.3
Bruxism is a common occurrence in the population (8%) and has been associated with alterations in the autonomic nervous system and stimulation of the TCR.2, 4, 5, 6 The mechanism behind the TCR stimulation is felt to be 2-fold. Firstly, masticatory movements (rhythmic masticatory muscle activity) and secondly, teeth contact can stimulate mechanoreceptors in the periodontal tissue.7 The link between bruxism, TCR, and alteration in the autonomic nervous system is important to highlight as it is well established that the autonomic nervous system plays a critical role in the pathogenesis of various cardiac arrhythmias, particularly atrial fibrillation. Although not specifically related to our patient, the fact that bruxism is so common raises the potential role it could be contributing to autonomic drivers of atrial fibrillation, and this is something that requires further research examination.
2. Schames S.E., Schames J., Schames M., Chagall-Gungur S.S. Sleep bruxism, an autonomic self-regulating response by triggering the trigeminal cardiac reflex. J Calif Dent Assoc. 2012;40:670–671. 674–676. [PubMed]
3. Arasho B., Sandu N., Spiriev T., Prabhakar H., Schaller B. Management of the trigeminocardiac reflex: facts and own experience. Neurol India. 2009;57:375–380. [PubMed]
4. Gastaldo E., Quatrale R., Graziani A., Eleopra R., Tugnoli V., Tola M.R., Granieri E. The excitability of the trigeminal motor system in sleep bruxism: a transcranial magnetic stimulation and brainstem reflex study. J Orofac Pain. 2006;20:145–155. [PubMed]
5. Chowdhury T., Bindu B., Singh G.P., Schaller B. Sleep disorders: is the trigemino-cardiac reflex a missing link? Front Neurol. 2017;8:63. [PubMed]
6. Sjoholm T.T., Piha S.J., Lehtinen I. Cardiovascular autonomic control is disturbed in nocturnal teethgrinders. Clin Physiol. 1995;15:349–354. [PubMed]
7. Okada Y., Kamijo Y., Okazaki K., Masuki S., Goto M., Nose H. Pressor responses to isometric biting are evoked by somatosensory receptors in periodontal tissue in humans. J Appl Physiol. 2009;107:531–539. [PubMed]
Of all of the problems that can be caused by disorders of the TMJ, eye strain or TMJ vision problems may seem to be the strangest. How can a joint that controls the jaw cause problems with your eyesight? Through muscles and nerves, your TMJ is wired to nearly everything else in your head, neck and face.
TMJ causes headaches, and headaches can, in turn, cause vision problems. Another factor is a nerve that is responsible for more than half of the total input to the brain: the trigeminal nerve. Understanding a bit about the trigeminal nerve will make it easy to see the connection between TMJ disorder and vision.
The Sphenopalatine Ganglion is on the Maxillary Division of the Trigeminal Nerve and it carries Autonomic nerves, both Sympathetic and Parasympathetic to the Trigeminal Nerve. These nerves travel throughout the trigeminal system and to the brain. A simple Sphenopalatine Ganglion Block can give Amazing, often almost immediate relief of symptoms. Dr Shapira teaches courses on blocking the Sphenopalatine Ganglion both nationally and internationally. He is one of the only doctors who teaches “Self Administration of SPG Blocks” with cotton tipped nasal cannulas.
SPG Blocks can treat Migraines, Chronic Daily Headaches and many painful conditions of the Ears, Eyes, Sinus and Nasal regions.
Your TMJ: It’s got a lot of nerve
These three nerve braches make the connection between the jaw and the eyes rather clear. TMJ pain is transmitted through these nerve pathways, and it can result in pain in other parts of the face. Many TMJ patients experience disturbances in vision.
Other common complaints are:
Retro-orbital Pain or pain behind the eyes
pressure behind the eyes
sensitivity to light, OFTEN SEVERE AND SOMETIMES MISTAKENLY THOUGHT TO BE MIGRAINE
floaters (small moving spots that you see in your field of vision)
FLASHING LIGHTS IN THE EYES MAY BE RETINAL DETATCHMENT! SEEK IMMEDIATE MEDICAL CARE WITH OPTHAMOLOGIST.
Many patients feel they have a vision problem because pain makes it difficult to read, especially small print or off a computer
Beware the domino effect
Our reaction to pain can often make the situation worse. When we feel the pain of a headache, jaw pain, neck pain or other pain related to TMJ, we unconsciously respond by positioning ourselves differently or using our muscles differently in an attempt to alleviate the pain.
Pain in the face or head can have a domino effect, with one issue leading to another through the trigeminal nerve or our own reactions to the pain.
Your TMJ is an important – and potentially problematic – part of your body.
The two joints lie on either side of the head between the mandible; the lower jaw, and the temporal bone at the base of the skull. It’s what allows our jaws to open and close. Because using our jaws is involved in two of the primary functions of our lives: eating and speaking, the TM joints are susceptible to issues.
A TMJ disorder can be caused suddenly by an injury to the head or face, but more often, it builds up gradually. It can be due to our behavior or habits.
Some common habitual causes of TMJ disorder are:
Eating hard foods frequently or taking oversized bites
People who sing or speak for a living may be more likely to encounter TMJ issues. Anyone can develop TMJ problems, and in order to get rid of the pain, vision problems, or other issues caused by TMJ, the TMJ problem itself must be eliminated.
The field of TMJ and orofacial pain covers a wide variety of problems and differential diagnosis of orofacial pain is important.
Get relief from TMJ Vision Problems
TMJ is best treated with non-invasive methods that do not permanently change the structure of the jaw or teeth. Pain relievers can help, and there are jaw and posture exercises that can bring relief to TMJ sufferers. If you are experiencing vision problems, headaches, jaw pain or facial pain, TMJ could be the cause.
To learn more about TMJ or to make a consultation appointment, contact our office.